CXCR3-mediated chemotaxis of human T cells is regulated by a Gi- and phospholipase C-dependent pathway and not via activation of MEK/p44/p42 MAPK nor Akt/PI-3 kinase

M.J. Smit; P.W. Verdijk; E.M.H. van der Raaij-Helmer; M. Navis; P Hensbergen; R. Leurs; C.P. Tensen

doi:10.1182/blood-2002-12-3945

CXCR3-mediated chemotaxis of human T cells is regulated by a Gi- and phospholipase C-dependent pathway and not via activation of MEK/p44/p42 MAPK nor Akt/PI-3 kinase

M.J. Smit
, P.W. Verdijk
, E.M.H. van der Raaij-Helmer
, M. Navis
, P Hensbergen
, R. Leurs
, C.P. Tensen

Research output: Contribution to Journal › Article › Academic › peer-review

Original language	English
Pages (from-to)	1959-65
Journal	Blood
Volume	102
Issue number	6
DOIs	https://doi.org/10.1182/blood-2002-12-3945
Publication status	Published - 2003

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

SDG 3 Good Health and Well-being

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10.1182/blood-2002-12-3945

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Cite this

@article{11fead47e5824bd7bf5b9cf27e22bf70,

title = "CXCR3-mediated chemotaxis of human T cells is regulated by a Gi- and phospholipase C-dependent pathway and not via activation of MEK/p44/p42 MAPK nor Akt/PI-3 kinase",

author = "M.J. Smit and P.W. Verdijk and \{van der Raaij-Helmer\}, E.M.H. and M. Navis and P Hensbergen and R. Leurs and C.P. Tensen",

year = "2003",

doi = "10.1182/blood-2002-12-3945",

language = "English",

volume = "102",

pages = "1959--65",

journal = "Blood",

issn = "0006-4971",

publisher = "Elsevier B.V.",

number = "6",

}

TY - JOUR

T1 - CXCR3-mediated chemotaxis of human T cells is regulated by a Gi- and phospholipase C-dependent pathway and not via activation of MEK/p44/p42 MAPK nor Akt/PI-3 kinase

AU - Smit, M.J.

AU - Verdijk, P.W.

AU - van der Raaij-Helmer, E.M.H.

AU - Navis, M.

AU - Hensbergen, P

AU - Leurs, R.

AU - Tensen, C.P.

PY - 2003

Y1 - 2003

U2 - 10.1182/blood-2002-12-3945

DO - 10.1182/blood-2002-12-3945

M3 - Article

SN - 0006-4971

VL - 102

SP - 1959

EP - 1965

JO - Blood

JF - Blood

IS - 6

ER -