Morphine causes a delayed increase in glutamate receptor functioning in the nucleus accumbens core

E.H. Jacobs; G. Wardeh; A.B. Smit; A.N.M. Schoffelmeer

doi:10.1016/j.ejphar.2005.02.009

Morphine causes a delayed increase in glutamate receptor functioning in the nucleus accumbens core

E.H. Jacobs, G. Wardeh, A.B. Smit, A.N.M. Schoffelmeer

Molecular and Cellular Neurobiology

Research output: Contribution to Journal › Article › Academic › peer-review

Abstract

Enhanced excitatory neurotransmission in the mesocorticolimbic system may contribute to the persistence of addiction behaviour. Here, we demonstrated that glutamate-, N-methyl-d-aspartate (NMDA)- and α-amino-3-hydroxy-5-methyl- 4-isoxazole propionic acid (AMPA)-induced [

Original language	English
Pages (from-to)	27-30
Number of pages	4
Journal	European Journal of Pharmacology
Volume	511
Issue number	1
DOIs	https://doi.org/10.1016/j.ejphar.2005.02.009
Publication status	Published - 2005

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10.1016/j.ejphar.2005.02.009

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title = "Morphine causes a delayed increase in glutamate receptor functioning in the nucleus accumbens core",

abstract = "Enhanced excitatory neurotransmission in the mesocorticolimbic system may contribute to the persistence of addiction behaviour. Here, we demonstrated that glutamate-, N-methyl-d-aspartate (NMDA)- and α-amino-3-hydroxy-5-methyl- 4-isoxazole propionic acid (AMPA)-induced [",

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AU - Wardeh, G.

AU - Smit, A.B.

AU - Schoffelmeer, A.N.M.

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AB - Enhanced excitatory neurotransmission in the mesocorticolimbic system may contribute to the persistence of addiction behaviour. Here, we demonstrated that glutamate-, N-methyl-d-aspartate (NMDA)- and α-amino-3-hydroxy-5-methyl- 4-isoxazole propionic acid (AMPA)-induced [

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DO - 10.1016/j.ejphar.2005.02.009

M3 - Article

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EP - 30

JO - European Journal of Pharmacology

JF - European Journal of Pharmacology

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Morphine causes a delayed increase in glutamate receptor functioning in the nucleus accumbens core

Abstract

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