Abstract
Much of Parkinson's research over the last decade has focused on cellular stress as a candidate mechanism. In this issue of Neuron, a new study by Matta et al. (2012) addressing the biological functions of the Parkinson's gene LRRK2 now identifies a presynaptic substrate, homing in on the idea that synapse loss might be a central early aspect of neurodegeneration. © 2012 Elsevier Inc.
Original language | English |
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Pages (from-to) | 935-938 |
Journal | Neuron |
Volume | 75 |
Issue number | 6 |
DOIs | |
Publication status | Published - 2012 |