Non-fibrillar beta-amyloid abates spike-timing-dependent synaptic potentiation at excitatory synapses in layer 2/3 of the neocortex by targeting postsynaptic AMPA receptors

I. Shemer, C. Holmgren, R. Min, L. Fülöp, M. Zilberter, K.M. Sousa, T. Farkas, W. Härtig, B. Penke, N. Burnashev, H. Tanila, Z. Zilberter, T. Harkany

Research output: Contribution to JournalArticleAcademicpeer-review

Abstract

Cognitive decline in Alzheimer's disease (AD) stems from the progressive dysfunction of synaptic connections within cortical neuronal microcircuits. Recently, soluble amyloid β protein oligomers (Aβ
Original languageEnglish
Pages (from-to)2035-2047
Number of pages13
JournalEuropean Journal of Neuroscience
Volume23
Issue number8
DOIs
Publication statusPublished - 2006

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