TY - JOUR
T1 - The nature of MIS 3 stadial-interstadial transitions in Europe: new insights from model-data comparisons
AU - van Meerbeeck, C.J.V.C.
AU - Renssen, H.
AU - Roche, D.M.V.A.P.
AU - Wohlfarth, B.
AU - Bohncke, S.J.P.
AU - Bos, J.A.A.
AU - Engels, S.
AU - Helmens, K.F.
AU - Sáncez-Goñi, M.F.
AU - Svensson, A.
AU - Vandenberghe, J.
PY - 2011
Y1 - 2011
N2 - 15 abrupt warming transitions perturbed glacial climate in Greenland during Marine Isotope Stage 3 (MIS 3, 60-27 ka BP). One hypothesis states that the 8-16 °C warming between Greenland Stadials (GS) and Interstadials (GI) was caused by enhanced heat transport to the North Atlantic region after a resumption of the Atlantic Meridional Overturning Circulation (AMOC) from a weak or shutdown stadial mode. This hypothesis also predicts warming over Europe, a prediction poorly constrained by data due to the paucity of well-dated quantitative temperature records. We therefore use a new evidence from biotic proxies and a climate model simulation to study the characteristics of a GS-GI transition in continental Europe and the link to enhanced AMOC strength. We compare reconstructed climatic and vegetation changes between a stadial and subsequent interstadial - correlated to GS15 and GI14 (∼55 ka BP) - with a simulated AMOC resumption using a three-dimensional earth system model setup with early-MIS 3 boundary conditions. Over western Europe (12°W-15°E), we simulate twice the annual precipitation, a 17 °C warmer coldest month, a 8 °C warmer warmest month, 1300 °C-day more growing degree days with baseline 5 °C (GDD5) and potential vegetation allowing tree cover after the transition. However, the combined effect of frequent killing frosts, <20 mm summer precipitation and too few GDD5 after the transition suggest a northern tree limit lying at ∼50°N during GI14. With these 3 climatic limiting factors we provide a possible explanation for the absence of forests north of 48°N during MIS 3 interstadials with mild summers. Finally, apart from a large model bias in warmest month surface air temperatures, our simulation is in reasonable agreement with reconstructed climatic and vegetation changes in Europe, thus further supporting the hypothesis. © 2011 Elsevier Ltd.
AB - 15 abrupt warming transitions perturbed glacial climate in Greenland during Marine Isotope Stage 3 (MIS 3, 60-27 ka BP). One hypothesis states that the 8-16 °C warming between Greenland Stadials (GS) and Interstadials (GI) was caused by enhanced heat transport to the North Atlantic region after a resumption of the Atlantic Meridional Overturning Circulation (AMOC) from a weak or shutdown stadial mode. This hypothesis also predicts warming over Europe, a prediction poorly constrained by data due to the paucity of well-dated quantitative temperature records. We therefore use a new evidence from biotic proxies and a climate model simulation to study the characteristics of a GS-GI transition in continental Europe and the link to enhanced AMOC strength. We compare reconstructed climatic and vegetation changes between a stadial and subsequent interstadial - correlated to GS15 and GI14 (∼55 ka BP) - with a simulated AMOC resumption using a three-dimensional earth system model setup with early-MIS 3 boundary conditions. Over western Europe (12°W-15°E), we simulate twice the annual precipitation, a 17 °C warmer coldest month, a 8 °C warmer warmest month, 1300 °C-day more growing degree days with baseline 5 °C (GDD5) and potential vegetation allowing tree cover after the transition. However, the combined effect of frequent killing frosts, <20 mm summer precipitation and too few GDD5 after the transition suggest a northern tree limit lying at ∼50°N during GI14. With these 3 climatic limiting factors we provide a possible explanation for the absence of forests north of 48°N during MIS 3 interstadials with mild summers. Finally, apart from a large model bias in warmest month surface air temperatures, our simulation is in reasonable agreement with reconstructed climatic and vegetation changes in Europe, thus further supporting the hypothesis. © 2011 Elsevier Ltd.
U2 - 10.1016/j.quascirev.2011.08.002
DO - 10.1016/j.quascirev.2011.08.002
M3 - Article
SN - 0277-3791
VL - 30
SP - 3618
EP - 3637
JO - Quaternary Science Reviews
JF - Quaternary Science Reviews
IS - 25-26
ER -